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GHK-Cu exerts its effects primarily through gene expression modulation, influencing approximately 31% of the human genome by upregulating genes involved in antioxidant responses, DNA repair, protein metabolism, and cellular growth while downregulating inflammatory mediators, fibrinogen synthesis, and pathological processes including cancer metastasis

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To investigate the mechanism underlying the effect of GSH on TGF signalling, we next detected TGF1 protein expression and SMAD3 phosphorylation

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[85] administered MSCs with atorvastatin or pravastatin at clinically appropriate levels and evaluated MSC generation, differentiation possibility, and gene expression profile